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The book contains nine articles on the subject of diagnosis and treatment of macular edema due to different causes. It presents both reviews of current recommendations of macular edema treatment as well as original research on specific forms of that entity.
Medicine --- angiogenesis --- angiopoietin-1 --- central serous chorioretinopathy --- proangiogenic factors --- combined treatment --- subthreshold diode micropulse --- anti-VEGF treatment --- diabetic macular edema --- retinal vein occlusion --- Coats disease --- peripheral retinal telangiectasia --- laser photocoagulation --- hypotony maculopathy --- complications of antiglaucoma surgery --- bleb compressive sutures --- age-related macular degeneration --- AMD --- proteomics --- tear film --- tear film proteome --- protein clearance --- neovascularization --- neovascular AMD --- macular edema --- uveitis --- uveitic complications --- non-steroidal anti-inflammatory drugs --- steroids --- biologic treatment --- Irvine–Gass syndrome --- cystoid macular edema --- pseudophakic cystoid macular edema --- NSAIDs corticosteroids --- anti-VEGF --- intravitreal injections --- intravitreal agents --- best corrected visual acuity --- central retinal thickness --- adverse drug reactions --- intravitreal drug expenses
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The book contains nine articles on the subject of diagnosis and treatment of macular edema due to different causes. It presents both reviews of current recommendations of macular edema treatment as well as original research on specific forms of that entity.
angiogenesis --- angiopoietin-1 --- central serous chorioretinopathy --- proangiogenic factors --- combined treatment --- subthreshold diode micropulse --- anti-VEGF treatment --- diabetic macular edema --- retinal vein occlusion --- Coats disease --- peripheral retinal telangiectasia --- laser photocoagulation --- hypotony maculopathy --- complications of antiglaucoma surgery --- bleb compressive sutures --- age-related macular degeneration --- AMD --- proteomics --- tear film --- tear film proteome --- protein clearance --- neovascularization --- neovascular AMD --- macular edema --- uveitis --- uveitic complications --- non-steroidal anti-inflammatory drugs --- steroids --- biologic treatment --- Irvine–Gass syndrome --- cystoid macular edema --- pseudophakic cystoid macular edema --- NSAIDs corticosteroids --- anti-VEGF --- intravitreal injections --- intravitreal agents --- best corrected visual acuity --- central retinal thickness --- adverse drug reactions --- intravitreal drug expenses
Choose an application
The book contains nine articles on the subject of diagnosis and treatment of macular edema due to different causes. It presents both reviews of current recommendations of macular edema treatment as well as original research on specific forms of that entity.
Medicine --- angiogenesis --- angiopoietin-1 --- central serous chorioretinopathy --- proangiogenic factors --- combined treatment --- subthreshold diode micropulse --- anti-VEGF treatment --- diabetic macular edema --- retinal vein occlusion --- Coats disease --- peripheral retinal telangiectasia --- laser photocoagulation --- hypotony maculopathy --- complications of antiglaucoma surgery --- bleb compressive sutures --- age-related macular degeneration --- AMD --- proteomics --- tear film --- tear film proteome --- protein clearance --- neovascularization --- neovascular AMD --- macular edema --- uveitis --- uveitic complications --- non-steroidal anti-inflammatory drugs --- steroids --- biologic treatment --- Irvine–Gass syndrome --- cystoid macular edema --- pseudophakic cystoid macular edema --- NSAIDs corticosteroids --- anti-VEGF --- intravitreal injections --- intravitreal agents --- best corrected visual acuity --- central retinal thickness --- adverse drug reactions --- intravitreal drug expenses --- angiogenesis --- angiopoietin-1 --- central serous chorioretinopathy --- proangiogenic factors --- combined treatment --- subthreshold diode micropulse --- anti-VEGF treatment --- diabetic macular edema --- retinal vein occlusion --- Coats disease --- peripheral retinal telangiectasia --- laser photocoagulation --- hypotony maculopathy --- complications of antiglaucoma surgery --- bleb compressive sutures --- age-related macular degeneration --- AMD --- proteomics --- tear film --- tear film proteome --- protein clearance --- neovascularization --- neovascular AMD --- macular edema --- uveitis --- uveitic complications --- non-steroidal anti-inflammatory drugs --- steroids --- biologic treatment --- Irvine–Gass syndrome --- cystoid macular edema --- pseudophakic cystoid macular edema --- NSAIDs corticosteroids --- anti-VEGF --- intravitreal injections --- intravitreal agents --- best corrected visual acuity --- central retinal thickness --- adverse drug reactions --- intravitreal drug expenses
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The combination of an increasing prevalence of diabetes and the aging of populations enables the appearance of a greater number of associated complications such as diabetic retinopathy. Diabetic retinopathy is the leading cause of preventable vision loss in working-age adults. The objective of this Special Issue is to highlight the existing evidence regarding the relationship between oxidative stress and low-grade chronic inflammation induced by hyperglycemia with the development and progression of diabetic retinopathy, with an emphasis on the importance of early diagnosis and the use of antioxidant and anti-inflammatory approaches to prevent or delay the harmful effects of diabetes on retinal tissue.
Medicine --- eicosanoids --- oxidative stress --- diabetic retinopathy --- cyclooxygenase --- lipoxygenase --- Cytochrome P450 --- HDAC6 --- tubastatin A --- retinal endothelial cells --- retinal endothelial cell senescence --- db/db mice --- Cinnamomi Ramulus --- Paeoniae Radix --- CPA4-1 --- blood-retinal barrier --- occludin --- human retina --- epiretinal membrane --- internal limiting membrane --- vitreoretinal surgery --- macular hole --- proliferative diabetic retinopathy --- antioxidants --- diabetes mellitus --- free radicals --- high-mobility group box 1 (HMGB1) --- inflammatory pathways --- novel therapies --- diabetic retinopathy (DR) --- inflammation --- angiogenesis --- extracellular vesicles --- miRNA --- biomarkers --- apoptosis --- fenofibrate --- thioredoxin --- hyperglycemia --- astaxanthin --- carotenoid --- reactive oxygen species --- photoreceptor cells --- PI3K --- Nrf2 --- eicosapentaenoic acid (EPA) --- docosahexaenoic acid (DHA) --- retinal pigment epithelium --- antioxidant --- ascorbic acid --- retinal disease --- vitamin D --- GLP-1 --- superoxide dismutase --- biomarkers of diabetic retinopathy --- metabolic memory --- tear film --- aqueous humor --- vitreous humor --- mitochondria --- redox --- photoreceptor --- glycation --- aging --- glyoxalase --- eicosanoids --- oxidative stress --- diabetic retinopathy --- cyclooxygenase --- lipoxygenase --- Cytochrome P450 --- HDAC6 --- tubastatin A --- retinal endothelial cells --- retinal endothelial cell senescence --- db/db mice --- Cinnamomi Ramulus --- Paeoniae Radix --- CPA4-1 --- blood-retinal barrier --- occludin --- human retina --- epiretinal membrane --- internal limiting membrane --- vitreoretinal surgery --- macular hole --- proliferative diabetic retinopathy --- antioxidants --- diabetes mellitus --- free radicals --- high-mobility group box 1 (HMGB1) --- inflammatory pathways --- novel therapies --- diabetic retinopathy (DR) --- inflammation --- angiogenesis --- extracellular vesicles --- miRNA --- biomarkers --- apoptosis --- fenofibrate --- thioredoxin --- hyperglycemia --- astaxanthin --- carotenoid --- reactive oxygen species --- photoreceptor cells --- PI3K --- Nrf2 --- eicosapentaenoic acid (EPA) --- docosahexaenoic acid (DHA) --- retinal pigment epithelium --- antioxidant --- ascorbic acid --- retinal disease --- vitamin D --- GLP-1 --- superoxide dismutase --- biomarkers of diabetic retinopathy --- metabolic memory --- tear film --- aqueous humor --- vitreous humor --- mitochondria --- redox --- photoreceptor --- glycation --- aging --- glyoxalase
Choose an application
The combination of an increasing prevalence of diabetes and the aging of populations enables the appearance of a greater number of associated complications such as diabetic retinopathy. Diabetic retinopathy is the leading cause of preventable vision loss in working-age adults. The objective of this Special Issue is to highlight the existing evidence regarding the relationship between oxidative stress and low-grade chronic inflammation induced by hyperglycemia with the development and progression of diabetic retinopathy, with an emphasis on the importance of early diagnosis and the use of antioxidant and anti-inflammatory approaches to prevent or delay the harmful effects of diabetes on retinal tissue.
Medicine --- eicosanoids --- oxidative stress --- diabetic retinopathy --- cyclooxygenase --- lipoxygenase --- Cytochrome P450 --- HDAC6 --- tubastatin A --- retinal endothelial cells --- retinal endothelial cell senescence --- db/db mice --- Cinnamomi Ramulus --- Paeoniae Radix --- CPA4-1 --- blood-retinal barrier --- occludin --- human retina --- epiretinal membrane --- internal limiting membrane --- vitreoretinal surgery --- macular hole --- proliferative diabetic retinopathy --- antioxidants --- diabetes mellitus --- free radicals --- high-mobility group box 1 (HMGB1) --- inflammatory pathways --- novel therapies --- diabetic retinopathy (DR) --- inflammation --- angiogenesis --- extracellular vesicles --- miRNA --- biomarkers --- apoptosis --- fenofibrate --- thioredoxin --- hyperglycemia --- astaxanthin --- carotenoid --- reactive oxygen species --- photoreceptor cells --- PI3K --- Nrf2 --- eicosapentaenoic acid (EPA) --- docosahexaenoic acid (DHA) --- retinal pigment epithelium --- antioxidant --- ascorbic acid --- retinal disease --- vitamin D --- GLP-1 --- superoxide dismutase --- biomarkers of diabetic retinopathy --- metabolic memory --- tear film --- aqueous humor --- vitreous humor --- mitochondria --- redox --- photoreceptor --- glycation --- aging --- glyoxalase --- n/a
Choose an application
The combination of an increasing prevalence of diabetes and the aging of populations enables the appearance of a greater number of associated complications such as diabetic retinopathy. Diabetic retinopathy is the leading cause of preventable vision loss in working-age adults. The objective of this Special Issue is to highlight the existing evidence regarding the relationship between oxidative stress and low-grade chronic inflammation induced by hyperglycemia with the development and progression of diabetic retinopathy, with an emphasis on the importance of early diagnosis and the use of antioxidant and anti-inflammatory approaches to prevent or delay the harmful effects of diabetes on retinal tissue.
eicosanoids --- oxidative stress --- diabetic retinopathy --- cyclooxygenase --- lipoxygenase --- Cytochrome P450 --- HDAC6 --- tubastatin A --- retinal endothelial cells --- retinal endothelial cell senescence --- db/db mice --- Cinnamomi Ramulus --- Paeoniae Radix --- CPA4-1 --- blood-retinal barrier --- occludin --- human retina --- epiretinal membrane --- internal limiting membrane --- vitreoretinal surgery --- macular hole --- proliferative diabetic retinopathy --- antioxidants --- diabetes mellitus --- free radicals --- high-mobility group box 1 (HMGB1) --- inflammatory pathways --- novel therapies --- diabetic retinopathy (DR) --- inflammation --- angiogenesis --- extracellular vesicles --- miRNA --- biomarkers --- apoptosis --- fenofibrate --- thioredoxin --- hyperglycemia --- astaxanthin --- carotenoid --- reactive oxygen species --- photoreceptor cells --- PI3K --- Nrf2 --- eicosapentaenoic acid (EPA) --- docosahexaenoic acid (DHA) --- retinal pigment epithelium --- antioxidant --- ascorbic acid --- retinal disease --- vitamin D --- GLP-1 --- superoxide dismutase --- biomarkers of diabetic retinopathy --- metabolic memory --- tear film --- aqueous humor --- vitreous humor --- mitochondria --- redox --- photoreceptor --- glycation --- aging --- glyoxalase --- n/a
Listing 1 - 6 of 6 |
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