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Energy metabolism at whole-body, cellular, and even organelle, i.e., mitochondrial, levels requires adequate regulation in order to maintain or improve (metabolic) health. In eukaryotic cells, mitochondria are key players in energy (ATP) production via oxidative phosphorylation. Both macro- and micronutrients potentially influence energy metabolism and mitochondrial functioning, either as substrates for (oxidative) catabolism or as essential constituents of enzymes or protein complexes involved in (mitochondrial) energy metabolism. This book contains a valuable collection of empirical preclinical and human studies to assist in the development of understanding and progress this area of research on improving health and, more specifically, metabolic health.
Medicine --- insulin --- lipogenesis --- obesity --- glucose homeostasis --- adipocytes --- cortisol --- ketones --- s-IgA --- exercise --- low carbohydrate diet --- NAFLD --- omega-3 --- krill oil --- phospholipids --- high-fat diet --- C57BL/6N mice --- thermoneutral temperature --- iron deficiency --- striated skeletal muscle --- physical capacity --- fatigue --- mitochondrial metabolism --- complex I --- resting metabolic rate --- prediction equation --- ageing adults --- overweight --- adiposity-based chronic disease --- energy balance --- metabolism --- insulin --- lipogenesis --- obesity --- glucose homeostasis --- adipocytes --- cortisol --- ketones --- s-IgA --- exercise --- low carbohydrate diet --- NAFLD --- omega-3 --- krill oil --- phospholipids --- high-fat diet --- C57BL/6N mice --- thermoneutral temperature --- iron deficiency --- striated skeletal muscle --- physical capacity --- fatigue --- mitochondrial metabolism --- complex I --- resting metabolic rate --- prediction equation --- ageing adults --- overweight --- adiposity-based chronic disease --- energy balance --- metabolism
Choose an application
Energy metabolism at whole-body, cellular, and even organelle, i.e., mitochondrial, levels requires adequate regulation in order to maintain or improve (metabolic) health. In eukaryotic cells, mitochondria are key players in energy (ATP) production via oxidative phosphorylation. Both macro- and micronutrients potentially influence energy metabolism and mitochondrial functioning, either as substrates for (oxidative) catabolism or as essential constituents of enzymes or protein complexes involved in (mitochondrial) energy metabolism. This book contains a valuable collection of empirical preclinical and human studies to assist in the development of understanding and progress this area of research on improving health and, more specifically, metabolic health.
Medicine --- insulin --- lipogenesis --- obesity --- glucose homeostasis --- adipocytes --- cortisol --- ketones --- s-IgA --- exercise --- low carbohydrate diet --- NAFLD --- omega-3 --- krill oil --- phospholipids --- high-fat diet --- C57BL/6N mice --- thermoneutral temperature --- iron deficiency --- striated skeletal muscle --- physical capacity --- fatigue --- mitochondrial metabolism --- complex I --- resting metabolic rate --- prediction equation --- ageing adults --- overweight --- adiposity-based chronic disease --- energy balance --- metabolism --- n/a
Choose an application
Energy metabolism at whole-body, cellular, and even organelle, i.e., mitochondrial, levels requires adequate regulation in order to maintain or improve (metabolic) health. In eukaryotic cells, mitochondria are key players in energy (ATP) production via oxidative phosphorylation. Both macro- and micronutrients potentially influence energy metabolism and mitochondrial functioning, either as substrates for (oxidative) catabolism or as essential constituents of enzymes or protein complexes involved in (mitochondrial) energy metabolism. This book contains a valuable collection of empirical preclinical and human studies to assist in the development of understanding and progress this area of research on improving health and, more specifically, metabolic health.
insulin --- lipogenesis --- obesity --- glucose homeostasis --- adipocytes --- cortisol --- ketones --- s-IgA --- exercise --- low carbohydrate diet --- NAFLD --- omega-3 --- krill oil --- phospholipids --- high-fat diet --- C57BL/6N mice --- thermoneutral temperature --- iron deficiency --- striated skeletal muscle --- physical capacity --- fatigue --- mitochondrial metabolism --- complex I --- resting metabolic rate --- prediction equation --- ageing adults --- overweight --- adiposity-based chronic disease --- energy balance --- metabolism --- n/a
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