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Brucellosis, caused by the facultative intracellular bacteria Brucella species, is one the most prevalent zoonosis worldwide. • The articles described in this book report several aspects of host-Brucella interactions. • The findings described here will help to advance in the comprehension of bacterial pathogenesis and contribute to the future development of drugs or vaccines to control brucellosis.

Research & information: general --- Recombinant vaccine --- divalent vaccine --- brucellosis --- Omp25 --- L7/L12 --- Brucella abortus 544 --- ST2 receptor --- Brucella abortus --- oral infection --- human endometrial cells --- internalization --- intracellular replication --- decidualization --- chemokines --- macrophages --- Brucella --- HSC --- MHC --- IL-10 --- cell cycle --- (p)ppGpp --- rsh --- neurobrucellosis --- platelets --- brain microvascular endothelial cells --- endothelial cells --- adhesins --- Ig-like domain --- monomeric autotransporters --- trimeric autotransporters --- extracellular matrix --- polar localization --- virulence factors --- vaccine candidates --- fibronectin --- canonical inflammasome --- non-canonical inflammasome --- NLR --- pyroptosis --- ASC --- caspase-11 --- caspase-1 --- IL-1β --- gDNA --- replication niche --- reservoir --- persistence --- survival --- chronic infection --- n/a

Choose an application

• Reference Manager
• EndNote
• RefWorks (Direct export to RefWorks)

Brucellosis, caused by the facultative intracellular bacteria Brucella species, is one the most prevalent zoonosis worldwide. • The articles described in this book report several aspects of host-Brucella interactions. • The findings described here will help to advance in the comprehension of bacterial pathogenesis and contribute to the future development of drugs or vaccines to control brucellosis.

Research & information: general --- Recombinant vaccine --- divalent vaccine --- brucellosis --- Omp25 --- L7/L12 --- Brucella abortus 544 --- ST2 receptor --- Brucella abortus --- oral infection --- human endometrial cells --- internalization --- intracellular replication --- decidualization --- chemokines --- macrophages --- Brucella --- HSC --- MHC --- IL-10 --- cell cycle --- (p)ppGpp --- rsh --- neurobrucellosis --- platelets --- brain microvascular endothelial cells --- endothelial cells --- adhesins --- Ig-like domain --- monomeric autotransporters --- trimeric autotransporters --- extracellular matrix --- polar localization --- virulence factors --- vaccine candidates --- fibronectin --- canonical inflammasome --- non-canonical inflammasome --- NLR --- pyroptosis --- ASC --- caspase-11 --- caspase-1 --- IL-1β --- gDNA --- replication niche --- reservoir --- persistence --- survival --- chronic infection