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The small phenolic compound salicylic acid (SA) is critical for plant defense against a broad spectrum of pathogens. SA is also involved in multi-layered defense responses, from pathogen-associated molecular pattern triggered basal defense, resistance gene-mediated defense, to systemic acquired resistance. Recent decades have witnessed tremendous progress towards our understanding of SA-mediated signaling networks. Many genes have been identified to have direct or indirect effect on SA biosynthesis or to regulate SA accumulation. Several SA receptors have been identified and characterization of these receptors has shed light on the mechanisms of SA-mediated defense signaling, which encompass chromosomal remodeling, DNA repair, epigenetics, to transcriptional reprogramming. Molecules from plant-associated microbes have been identified, which manipulate SA levels and/or SA signaling. SA does not act alone. It engages in crosstalk with other signaling pathways, such as those mediated by other phytohormones, in an agonistic or antagonistic manner, depending on hormones and pathosystems. Besides affecting plant innate immunity, SA has also been implicated in other cellular processes, such as flowering time determination, lipid metabolism, circadian clock control, and abiotic stress responses, possibly contributing to the regulation of plant development. The multifaceted function of SA makes it critically important to further identify genes involved in SA signaling networks, understand their modes of action, and delineate interactions among the components of SA signaling networks. In addition, genetic manipulation of genes involved in SA signaling networks has also provided a promising approach to enhance disease resistance in economically important plants. This ebook collects articles in the Research Topic "Salicylic Acid Signaling Networks". For this collection we solicited reviews, perspectives, and original research articles that highlight recent exciting progress on the understanding of molecular mechanisms underlying SA-mediated defense, SA-crosstalk with other pathways and how microbes impact these events.

Circadian clock --- systemic acquired resistance --- Reactive Oxygen Species --- crosstalk --- pathogen effector --- NPR1 --- Cellular redox --- Lipid Metabolism --- flowering --- SA receptors --- Circadian clock --- systemic acquired resistance --- Reactive Oxygen Species --- crosstalk --- pathogen effector --- NPR1 --- Cellular redox --- Lipid Metabolism --- flowering --- SA receptors

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This eBook is a collection of articles from a Frontiers Research Topic. Frontiers Research Topics are very popular trademarks of the Frontiers Journals Series: they are collections of at least ten articles, all centered on a particular subject. With their unique mix of varied contributions from Original Research to Review Articles, Frontiers Research Topics unify the most influential researchers, the latest key findings and historical advances in a hot research area! Find out more on how to host your own Frontiers Research Topic or contribute to one as an author by contacting the Frontiers Editorial Office: frontiersin.org/about/contact

Science: general issues --- Pharmacology --- immune checkpoint --- cancer therapy --- gene regulation --- acquired resistance --- CAR-T and CAR-NK cell-therapy

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The small phenolic compound salicylic acid (SA) is critical for plant defense against a broad spectrum of pathogens. SA is also involved in multi-layered defense responses, from pathogen-associated molecular pattern triggered basal defense, resistance gene-mediated defense, to systemic acquired resistance. Recent decades have witnessed tremendous progress towards our understanding of SA-mediated signaling networks. Many genes have been identified to have direct or indirect effect on SA biosynthesis or to regulate SA accumulation. Several SA receptors have been identified and characterization of these receptors has shed light on the mechanisms of SA-mediated defense signaling, which encompass chromosomal remodeling, DNA repair, epigenetics, to transcriptional reprogramming. Molecules from plant-associated microbes have been identified, which manipulate SA levels and/or SA signaling. SA does not act alone. It engages in crosstalk with other signaling pathways, such as those mediated by other phytohormones, in an agonistic or antagonistic manner, depending on hormones and pathosystems. Besides affecting plant innate immunity, SA has also been implicated in other cellular processes, such as flowering time determination, lipid metabolism, circadian clock control, and abiotic stress responses, possibly contributing to the regulation of plant development. The multifaceted function of SA makes it critically important to further identify genes involved in SA signaling networks, understand their modes of action, and delineate interactions among the components of SA signaling networks. In addition, genetic manipulation of genes involved in SA signaling networks has also provided a promising approach to enhance disease resistance in economically important plants. This ebook collects articles in the Research Topic "Salicylic Acid Signaling Networks". For this collection we solicited reviews, perspectives, and original research articles that highlight recent exciting progress on the understanding of molecular mechanisms underlying SA-mediated defense, SA-crosstalk with other pathways and how microbes impact these events.

Circadian clock --- systemic acquired resistance --- Reactive Oxygen Species --- crosstalk --- pathogen effector --- NPR1 --- Cellular redox --- Lipid Metabolism --- flowering --- SA receptors

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This book was established after closing the Special Issue “Decline of Mediterranean Fruit Crops and Forests Associated with Fungal Trunk Pathogens” edited by Dr. Carlos Agustí-Brisach as Guest Editor and Mr. Everett Zhu as Manager Editor. This book represents a collection of papers related to the etiology, epidemiology, and control of fungal trunk diseases in several Mediterranean woody crops such as almond, citrus, grapevine or pistachio, among others. This volume was intended to generate knowledge on the etiology and epidemiology of tree decline syndromes to build a strong foundation for developing effective management approaches to reduce the yield losses caused by these diseases. In addition, a broad collection of techniques to conduct studies on etiology and epidemiology as well as to determine the effect of biotic and abiotic factors enhancing the progression of trunk diseases in woody crops are shown in this book.

Research & information: general --- Biology, life sciences --- Technology, engineering, agriculture --- grapevine trunk diseases --- signal transduction --- systemic acquired resistance --- preformed defenses --- symptom severity --- pistachio --- crown rot --- root rot --- Macrophomina phaseolina --- pathogenicity --- bscogniauxia --- botryosphaeriaceae --- cadophora --- citrus dieback --- colletotrichum --- phaeoacremonium --- fungal trunk pathogens --- occurrence --- Prunus dulcis --- water deficiency --- weakened hosts --- grapevine --- Diplodia seriata --- Neofusicoccum parvum --- diagnostic --- Vitis vinifera --- Botryosphaeria dieback --- foliar symptoms --- (R)-mellein --- qPCR --- LC-MS/MS --- esca complex --- wood disease control --- leaf symptoms --- fungal pathogens --- natural infections --- Silverleaf disease --- Chondrostereum purpureum --- Prunus salicina --- Japanese plum --- apple die-back --- canker --- fungi --- multi-loci phylogeny --- wood decay --- grapevine trunk diseases --- signal transduction --- systemic acquired resistance --- preformed defenses --- symptom severity --- pistachio --- crown rot --- root rot --- Macrophomina phaseolina --- pathogenicity --- bscogniauxia --- botryosphaeriaceae --- cadophora --- citrus dieback --- colletotrichum --- phaeoacremonium --- fungal trunk pathogens --- occurrence --- Prunus dulcis --- water deficiency --- weakened hosts --- grapevine --- Diplodia seriata --- Neofusicoccum parvum --- diagnostic --- Vitis vinifera --- Botryosphaeria dieback --- foliar symptoms --- (R)-mellein --- qPCR --- LC-MS/MS --- esca complex --- wood disease control --- leaf symptoms --- fungal pathogens --- natural infections --- Silverleaf disease --- Chondrostereum purpureum --- Prunus salicina --- Japanese plum --- apple die-back --- canker --- fungi --- multi-loci phylogeny --- wood decay

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This open access book offers the first comprehensive account of the pan-genome concept and its manifold implications. The realization that the genetic repertoire of a biological species always encompasses more than the genome of each individual is one of the earliest examples of big data in biology that opened biology to the unbounded. The study of genetic variation observed within a species challenges existing views and has profound consequences for our understanding of the fundamental mechanisms underpinning bacterial biology and evolution. The underlying rationale extends well beyond the initial prokaryotic focus to all kingdoms of life and evolves into similar concepts for metagenomes, phenomes and epigenomes. The book’s respective chapters address a range of topics, from the serendipitous emergence of the pan-genome concept and its impacts on the fields of microbiology, vaccinology and antimicrobial resistance, to the study of microbial communities, bioinformatic applications and mathematical models that tie in with complex systems and economic theory. Given its scope, the book will appeal to a broad readership interested in population dynamics, evolutionary biology and genomics.

Microbial genetics. --- Microbial genomics. --- Evolutionary biology. --- Biomathematics. --- Microbial ecology. --- Human genetics. --- Genetics. --- Microbial Genetics and Genomics. --- Evolutionary Biology. --- Genetics and Population Dynamics. --- Microbial Ecology. --- Human Genetics. --- Genetics and Genomics. --- Biology --- Embryology --- Mendel's law --- Adaptation (Biology) --- Breeding --- Chromosomes --- Heredity --- Mutation (Biology) --- Variation (Biology) --- Genetics --- Heredity, Human --- Human biology --- Physical anthropology --- Environmental microbiology --- Microorganisms --- Ecology --- Microbiology --- Mathematics --- Animal evolution --- Animals --- Biological evolution --- Darwinism --- Evolutionary biology --- Evolutionary science --- Origin of species --- Evolution --- Biological fitness --- Homoplasy --- Natural selection --- Phylogeny --- Genomics --- Microbial genetics --- Genomes --- Biologia molecular --- Biofísica molecular --- Bioquímica molecular --- Biofísica --- Bioquímica --- Histoquímica --- Biologia molecular vegetal --- Codi genètic --- Diagnòstic molecular --- Endocrinologia molecular --- Evolució molecular --- Farmacologia molecular --- Genètica molecular --- Glicòmica --- Metabolòmica --- Microbiologia molecular --- Neurobiologia molecular --- Patologia molecular --- Proteòmica --- Reconeixement molecular --- Biomolècules --- Genoma --- Genoma humà --- Genòmica --- Genomes. --- Genomics. --- Genome research --- Molecular genetics --- Haploidy --- Research --- Microbial Genetics and Genomics --- Evolutionary Biology --- Genetics and Population Dynamics --- Microbial Ecology --- Human Genetics --- Genetics and Genomics --- Comparative genomics --- Metagenomics --- Microbial Population Analysis --- Pangenome Profile --- Supra-Genome Analysis --- Adaptive Evolution --- Computational Tools --- Bioinformatic Genomics --- Core Dispensable Genome --- Selection, Recombination, Composition --- Acquired Resistance --- Bacterial Species Concept --- Genomic Diversity --- Bacterial Ecology, Microevolution --- Open Access --- Pan-metagenomics --- Pan-microbiomics --- Pan-epigenome --- Gene Transfer --- Pan-phenomes --- Microbiology (non-medical) --- Genetics (non-medical) --- Applied mathematics --- Ecological science, the Biosphere --- Medical genetics