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Proefschriften --- Thèses --- Eau usée --- Wastewater --- Nitrate --- Nitrates --- Nitrite --- Nitrites --- Nitrification --- Gaz --- Gases --- Ammoniac --- Ammonia --- pH --- Température --- Temperature --- Biomasse --- biomass --- Oxydation --- Oxidation --- Traitement des eaux usées --- Wastewater treatment --- Réaction chimique --- chemical reactions --- Modèle --- Models --- Sciences and engineering --- physical sciences --- applied sciences --- engineering --- environmental --- Theses --- environmental. --- Temperature. --- Anammox --- Physical sciences --- Applied sciences --- Engineering --- Environmental.

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Pharmacology --- Nitrates --- Biological Science Disciplines --- Pharmaceutical Preparations --- Nitroglycerin. --- Nitrates. --- Nitrites. --- Anginine --- Dynamite --- Gilustenon --- Nitrangin --- Nitro-Bid --- Nitro-Dur --- Nitrocard --- Nitroderm --- Nitroderm TTS --- Nitroglyn --- Nitrol --- Nitrolan --- Nitrong --- Nitrospan --- Nitrostat --- Perlinganit --- Susadrin --- Sustac --- Sustak --- Sustonit --- Transderm Nitro --- Tridil --- Trinitrin --- Trinitrolong --- Glyceryl Trinitrate --- Nitro Bid --- Nitro Dur --- NitroBid --- NitroDur --- Trinitrate, Glyceryl --- Glyceryl trinitate --- Nitrites --- Glyceryl trinitate. --- Nitrite --- Nitrate --- Nitroglycerin

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Periodontitis is an infection-induced inflammatory disease accounting for huge healthcare costs and socio-economic impacts. Bacteria from the indigenous oral flora colonize the interspace between the tooth and the connective tissue, which induces an inflammatory response. If the bacteria proliferate and release virulence factors, they cause an imbalance in the host inflammatory response that induces degenerative processes in the surrounding tissues. This process is often slow, and the disease affects mainly older people, but the process could be rapid and affect young individuals if certain pathogens colonize the area. The two most studied periodontal pathogens, Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans, express virulence factors, including proteases and exotoxins. Periodontal bacteria and their products can be translocated to the peripheral circulation and are therefore linked to the risk pattern of several systemic diseases. However, it is not known if the increased risk for systemic disease associated with periodontitis is an effect of the invading bacteria and/or their released products, the release of components from the local inflammatory response, or a common host susceptibility pattern. The most studied periodontitis-associated systemic diseases are cardiovascular diseases and rheumatoid arthritis. Here, we want to shed light on mechanisms behind the associations of periodontal infections with systemic inflammation.

Medicine --- apical periodontitis --- adaptive immunity --- saliva --- serum --- antibody --- Aggregatibacter actinomycetemcomitans --- invasiveness --- leukotoxin --- cytolethal distending toxin --- serum resistance --- outer membrane vesicles --- oral microbiome --- nitric oxide --- nitrate --- nitrite --- periodontal disease --- alveolar bone loss --- gingiva --- bacteria --- biofilm --- immunity --- inflammation --- smoking --- Rheumatoid arthritis --- Porphyromonas gingivalis --- periodontitis --- citrullination --- peptidylarginine deiminase --- ACPA --- anti-CCP --- host response --- infection --- oral microbiota --- virulence factors --- metabolites --- TREM-1 --- intervention --- LP17 --- IL-17 --- RANKL --- OPG --- cardiovascular diseases --- rheumatoid arthritis --- inflammatory response --- apical periodontitis --- adaptive immunity --- saliva --- serum --- antibody --- Aggregatibacter actinomycetemcomitans --- invasiveness --- leukotoxin --- cytolethal distending toxin --- serum resistance --- outer membrane vesicles --- oral microbiome --- nitric oxide --- nitrate --- nitrite --- periodontal disease --- alveolar bone loss --- gingiva --- bacteria --- biofilm --- immunity --- inflammation --- smoking --- Rheumatoid arthritis --- Porphyromonas gingivalis --- periodontitis --- citrullination --- peptidylarginine deiminase --- ACPA --- anti-CCP --- host response --- infection --- oral microbiota --- virulence factors --- metabolites --- TREM-1 --- intervention --- LP17 --- IL-17 --- RANKL --- OPG --- cardiovascular diseases --- rheumatoid arthritis --- inflammatory response

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Periodontitis is an infection-induced inflammatory disease accounting for huge healthcare costs and socio-economic impacts. Bacteria from the indigenous oral flora colonize the interspace between the tooth and the connective tissue, which induces an inflammatory response. If the bacteria proliferate and release virulence factors, they cause an imbalance in the host inflammatory response that induces degenerative processes in the surrounding tissues. This process is often slow, and the disease affects mainly older people, but the process could be rapid and affect young individuals if certain pathogens colonize the area. The two most studied periodontal pathogens, Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans, express virulence factors, including proteases and exotoxins. Periodontal bacteria and their products can be translocated to the peripheral circulation and are therefore linked to the risk pattern of several systemic diseases. However, it is not known if the increased risk for systemic disease associated with periodontitis is an effect of the invading bacteria and/or their released products, the release of components from the local inflammatory response, or a common host susceptibility pattern. The most studied periodontitis-associated systemic diseases are cardiovascular diseases and rheumatoid arthritis. Here, we want to shed light on mechanisms behind the associations of periodontal infections with systemic inflammation.

apical periodontitis --- adaptive immunity --- saliva --- serum --- antibody --- Aggregatibacter actinomycetemcomitans --- invasiveness --- leukotoxin --- cytolethal distending toxin --- serum resistance --- outer membrane vesicles --- oral microbiome --- nitric oxide --- nitrate --- nitrite --- periodontal disease --- alveolar bone loss --- gingiva --- bacteria --- biofilm --- immunity --- inflammation --- smoking --- Rheumatoid arthritis --- Porphyromonas gingivalis --- periodontitis --- citrullination --- peptidylarginine deiminase --- ACPA --- anti-CCP --- host response --- infection --- oral microbiota --- virulence factors --- metabolites --- TREM-1 --- intervention --- LP17 --- IL-17 --- RANKL --- OPG --- cardiovascular diseases --- rheumatoid arthritis --- inflammatory response

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Hypertension is a major health problem worldwide, increasing cardiovascular (CV) risk and mortality. Together with pharmacological treatments, non-pharmacological approaches, such as nutrient intake modifications, play an important role in optimizing treatment. A link has been demonstrated between hypertension and body weight as well as dietary habits. The aim of this Special Issue is to improve the understanding of the relationships between some nutrients and hypertension, and of the effects of different dietary approaches on hypertension regulation from different points of view.

arterial stiffness --- rest raw material --- n/a --- Ojeoksan --- adhesion molecule --- cardiovascular risk factors --- renin-angiotensin-aldosterone system --- menopause --- children --- Mediterranean Diet --- fat --- salt intake --- renal sympathetic nerve activity --- obesity --- atherosclerosis --- parathyroid function --- endothelial function --- blood pressure --- fish protein --- nitric oxide --- l-NAME --- pregnancy --- weight loss --- magnesium --- polyphenol --- sodium --- nutrition --- nitrite --- developmental programming --- potassium --- sodium intake --- elderly --- amino acids --- pulse wave velocity --- calcium intake --- electrolytes --- hesperidin --- salt-sensitivity --- cardiovascular remodeling --- inflammation --- vascular inflammation --- diet --- renal transporters --- tea secondary metabolites --- endothelium --- calcium --- vasodilation --- vitamin D --- meta-analysis --- fructose --- sympathetic activity --- physical activity --- reprogramming --- fish meal --- Post Exercise Hypotension --- high blood pressure --- cod --- hypertension --- humans --- oxidative stress