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Age. --- Behavior. --- Infantile stimulation. --- Stimulation.
Article
Year: 1964
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Critical period. --- Emotional reactivity. --- Infantile stimulation. --- Periods. --- Reactivity. --- Stimulation. --- Stimulus. --- Theory.
Article
Year: 1991
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Avoidance. --- Behavior. --- Benzodiazepine receptor. --- Benzodiazepine. --- Blockade. --- Coping. --- Infantile stimulation. --- Perinatal. --- Rat. --- Rats. --- Receptor. --- Receptors. --- Stimulation.
Article
Year: 1992
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Acquisition. --- Adult. --- Avoidance. --- Behavior. --- Benzodiazepine receptor. --- Benzodiazepine. --- Coping. --- Emotion. --- Handling. --- Infantile stimulation. --- Rat. --- Receptor. --- Stimulation. --- System.
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Male Purdue-Wistar rats were handled for 20 days in infancy or were not disturbed (total N = 324). In adulthood these animals were subdivided and exposed to one of three electric shock intensities (0.2, 0.5, or 0.8 mA) for durations of 2, 5, 10, 15, 30, or 60 min. Following the termination of shock, the animals were killed immediately, 5 min afterwards, or 15 min afterwards; and the free plasma corticosterone was assayed. Both duration of shock and time between shock termination and decapitation affected the magnitude of the corticoid response; these effects were additive and permit the general conclusion that the major parameter is total time between shock onset and killing. Rats handled in infancy were found to have a greater amount of plasma corticosterone at the time that shock was terminated, but their rate of release of corticosterone during the subsequent 15 min was less than that of nonhandled controls.
Adulthood. --- Animal. --- Animals. --- Control. --- Corticosterone response. --- Corticosterone. --- Decapitation. --- Development. --- Duration. --- Electric shock. --- Function. --- Infantile stimulation. --- Male. --- Parameters. --- Plasma corticosterone. --- Plasma-corticosterone. --- Plasma. --- Rat. --- Rats. --- Release. --- Response. --- Shock. --- Stimulation. --- Stress. --- Time.
Article
Year: 2002
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Neonatal (early) handling (EH) and environmental enrichment (EE) of laboratory rodents have been the two most commonly used methods of providing supplementary environmental stimulation in order to study behavioral and neurobiological plasticity. A large body of research has been generated since the 1950s, unequivocally showing that both treatments induce profound and long-lasting behavioral and neural consequences while also inducing plastic brain effects and being "protective" against some age-related deficits. The present work is aimed at reviewing the main neurobehavioral effects of both manipulations, with the final purpose of comparing them and trying to find out to what extent the effects of both treatments may share (or not) possible neural mechanisms. (C) 2002 Published by Elsevier Science Inc
2-way active-avoidance. --- Adult-rats. --- Benzodiazepine receptor. --- Brain cortical membranes. --- Brain. --- Consequences. --- Deficits. --- Early experience,neonatal handling,environmental enrichment,stress,anxiety,learning,memory,neural plasticity,hippocampus,age-related deficits. --- Enrichment. --- Environmental enrichment. --- Handling. --- Infantile stimulation. --- Laboratory. --- Low-avoidance rats. --- Mechanisms. --- Method. --- Neonatal. --- Nerve growth-factor. --- Novelty-seeking. --- Perinatal flumazenil. --- Plasticity. --- Purpose. --- Research. --- Rodent. --- Rodents. --- Roman high-avoidance. --- Stimulation. --- Treatment. --- Work.
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