Choose an application

• Reference Manager
• EndNote
• RefWorks (Direct export to RefWorks)

"Brain aging has long been seen as a process of deterioration and decline. Today, this view been challenged with research showing that not all cognitive processes decline with age, that some improve over the course of adulthood, and those that improve can often compensate for those that decline. Chapters in this multidisciplinary volume examine the neural mechanisms underlying changes in the aging brain, changes in learning and memory, risk and protective factors, and the assessment and prevention of cognitive decline"--

Brain --- Cognition in old age. --- Cognitive Aging. --- Cognition. --- Cognitive Dysfunction --- Aging. --- physiology. --- prevention & control.

Choose an application

• Reference Manager
• EndNote
• RefWorks (Direct export to RefWorks)

Quatrième de couverture : "Les fonctions exécutives peuvent être définies par un rôle de contrôle intervenant dans les situations non familières ou complexes nécessitant la coordination de différentes actions vers un but finalisé. Il s'agit de capacités de contrôle de haut niveau impliquant la prise de décision et l'inhibition de comportements inappropriés. Les fonctions exécutives sont fréquemment altérées après un traumatisme crânien sévère, ce qui peut se traduire par des troubles cognitifs mais aussi par des troubles du comportement qui sont fréquents et peuvent représenter la gêne essentielle. Des modèles théoriques tentent d'expliquer ces fonctions exécutives. L'évaluation, en particulier écologique, est au centre de la prise en charge. Les approches rééducatives sont aujourd'hui multiples et découlent des approches théoriques avec des protocoles de restauration ou de réorganisation."

Personnes atteintes de lésions cérébrales --- Fonctions exécutives (neuropsychologie) --- Troubles du comportement --- Troubles de la cognition --- Remédiation cognitive --- Personnes atteintes de lésions cérébrales. --- Troubles du comportement. --- Troubles de la cognition. --- Remédiation cognitive. --- Brain Injuries, Traumatic --- Cognition Disorders --- Locomotion --- Cognitive Dysfunction

Choose an application

• Reference Manager
• EndNote
• RefWorks (Direct export to RefWorks)

This book provides up-to-date research on the clinical assessment and management of individuals with dementia and those with mild cognitive impairment (MCI), the fastest growing clinical population. A summary of important points at the end of chapters highlights essential clinical information and guides student learning. An all-new Clinical Guide comprises the second half of the book providing an extensive discussion of the process of assessment and evidence-based treatments for individuals in all stages of dementia.

Cognition disorders. --- Communicative disorders. --- Dementia. --- Aphrenia --- Aphronesia --- Athymia --- Dementias --- Brain --- Neurobehavioral disorders --- Psychoses --- Communication disorders (Medicine) --- Disorders of communication --- Nervous system --- Cognitive disorders --- Psychology, Pathological --- Diseases --- Cognition disorders --- Communicative disorders --- Dementia --- Cognitive Dysfunction --- Communication Disorders --- diagnosis --- etiology --- complications --- therapy

Choose an application

• Reference Manager
• EndNote
• RefWorks (Direct export to RefWorks)

1. 1. The classic animal models for human psychiatric conditions involves rodents As prey species, their normal behaviors of avoidance would be considered pathological in humans and dogs. Hence, such models may not be homologous for similar behaviors found in psychiatric pathology in humans. 2. 2. Dogs exhibit pathological behavioral conditions that may be equivalent to certain human psychiatric conditions. These canine conditions appear spontaneously or endogenously in the absence of genetic or neurochemcial manipulation, and as such, may be homologous to the human condition. 3. 3. If canine conditions approach homology with human conditions they should have excellent face, predictive, and construct validity. 4. 4. The canine conditions of separation anxiety, obsessive-compulsive disorder, cognitive dysfunction, dominance aggression, and panic disorder have good to excellent validity at all explored levels for human generalized anxiety disorder, obsessive-compulsive disorder, Alzheimer's disease, impulse control disorders, and panic disorder. 5. 5. Natural canine models can aid our understanding of human psychiatric conditions.

Absence. --- Aggression. --- Animal model. --- Animal-model. --- Animal-models. --- Animal. --- Anxiety. --- Assessment. --- Avoidance. --- Behavior. --- Canine model. --- Cognition. --- Cognitive disfunction dog model. --- Cognitive dysfunction. --- Control. --- Disease. --- Disorder. --- Dog. --- Dogs. --- Dominance aggression. --- Dominance. --- Generalized anxiety disorder. --- Genetic. --- Human. --- Humans. --- Impulse control disorder. --- Level. --- Model. --- Models. --- Natural. --- Obsessive-compulsive disorder. --- Obsessive-compulsive. --- Panic disorders. --- Panic. --- Post-traumatic stress syndrome. --- Prey. --- Rodent. --- Rodents. --- Schizophrenia. --- Separation anxiety. --- Separation.

Choose an application

• Reference Manager
• EndNote
• RefWorks (Direct export to RefWorks)

This book focuses on the recent advancements in both fundamental and clinical research, focusing on identifying, developing, and applying new and improved biological markers for specific neurologic disorders in the future. The original research work and review articles published here highlight some unique mechanisms underlying the most prevalent pathophysiological conditions affecting human health. Other areas covered in the book include emerging treatment options and correct diagnoses using different biochemical and imaging techniques.

Medicine --- Alzheimer’s disease --- amyloid beta --- hippocampus --- Kir channels --- K+ channels --- glioblastoma multiforme --- low-grade glioma --- astrocytoma --- recurrence --- multifocal growth --- mRNA expression --- MPS1 --- TTK --- therapy --- EDTA chelation therapy --- neurodegenerative diseases --- metal detoxification --- reactive oxygen species (ROS) --- neuroinflammation --- neurodegeneration --- cognitive dysfunction --- neurofibrillary tangles --- diagnosis methods --- biomarkers --- perinatal stress --- NURR1 --- inflammation --- late-onset diseases --- early life --- artificial neural network --- deep learning --- Parkinson’s disease --- atypical parkinsonian syndrome --- dopamine transporter SPECT --- β-Amyloid --- calpain --- Collapsin Response Mediator Protein-2 --- human-neuroblastoma SH-SY5Y cells --- Somatostatin-14 --- somatostatin receptor --- brain injury --- quinpirole --- dopamine D2 receptors --- glial cell --- autophagy --- mitophagy --- multiple sclerosis --- biomarker --- neurons --- astrocytes --- Alzheimer’s and Parkinson’s diseases --- fluid and imaging biomarkers --- amyloid-β and tau --- miRNA --- extracellular vesicles --- exosomes and ectosomes --- PET --- radiotracers --- radiolabeled molecules --- n/a --- Alzheimer's disease --- Parkinson's disease --- Alzheimer's and Parkinson's diseases