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Nitric oxide has proven to be a molecule with wide biological significance. It is involved in myriad actions which range from physiology to pathophysiology. One of the fundamental questions in relation to its biological relevance concerns the paradoxical nature of some of its actions. For example, there is a whole range of effects related to cytoprotection, cell proliferation, differentiation, and cell death. The way in which nitric oxide becomes involved in pathophysiology is slowly being elucidated. This volume contains a summary of the discussions that took place at the Second International Paraelios Symposium on Nitric Oxide research, and will make a significant contribution to this very exciting subject.Originally published in 1998.The Princeton Legacy Library uses the latest print-on-demand technology to again make available previously out-of-print books from the distinguished backlist of Princeton University Press. These editions preserve the original texts of these important books while presenting them in durable paperback and hardcover editions. The goal of the Princeton Legacy Library is to vastly increase access to the rich scholarly heritage found in the thousands of books published by Princeton University Press since its founding in 1905.

Nitric oxide --- Cell proliferation --- Cell differentiation --- Apoptosis --- Physiological effect --- Pathophysiology --- Molecular aspects --- 7-Chlorokynurenic acid. --- 7-Nitroindazole. --- AMPA receptor. --- AMPA. --- Aconitase. --- Activation. --- Alkaline phosphatase. --- Antioxidant. --- Apoptosis. --- Arginine. --- Aspartic acid. --- Bcl-2. --- Botulinum toxin. --- Carcinogenesis. --- Cell death. --- Cellular respiration. --- Chloral hydrate. --- Cyanide. --- Cyclooxygenase. --- Cyclothiazide. --- Cytochrome c oxidase. --- Cytostasis. --- Cytotoxicity. --- Dialysis. --- EGTA (chemical). --- Electron transport chain. --- Enantiomer. --- Endonuclease. --- Endothelial NOS. --- Ethylenediaminetetraacetic acid. --- Excitotoxicity. --- Exocytosis. --- Gliotoxin. --- Glomerulonephritis. --- Glutamic acid. --- Glutathione. --- Glyceraldehyde 3-phosphate dehydrogenase. --- Glycerol. --- Glycine. --- Hydrogen peroxide. --- IC50. --- Immortalised cell line. --- Immunoassay. --- Immunohistochemistry. --- Interferon. --- L1210 cells. --- Lipid peroxidation. --- Lipopolysaccharide. --- Lipoxygenase. --- Lysis. --- Macrophage. --- Methemoglobin. --- Mitochondrion. --- N-Methyl-D-aspartic acid. --- NMDA receptor. --- Necrosis. --- Nitration. --- Nitric oxide. --- Nitrite. --- Nitrosamine. --- Nitrosation. --- Nitroso. --- Nitrosylation. --- Nitrous oxide. --- Nitrovasodilator. --- Nocodazole. --- Norepinephrine. --- Oxidative stress. --- Oxide. --- Oxypurinol. --- P53. --- Penicillamine. --- Peroxide. --- Peroxynitrite. --- Phosphodiesterase inhibitor. --- Phosphodiesterase. --- Phospholipid. --- Pro-oxidant. --- Propidium iodide. --- Prostacyclin. --- Protonation. --- Pyruvic acid. --- Radical (chemistry). --- Reactive nitrogen species. --- Reuptake. --- S-Nitrosoglutathione. --- S-Nitrosylation. --- Sodium nitrite. --- Sodium nitroprusside. --- Somatostatin. --- Superoxide dismutase. --- Superoxide. --- T cell. --- TNF inhibitor. --- Tetanospasmin. --- Thromboxane A2. --- Transfection. --- Transition metal. --- Trifluoperazine. --- Xanthine oxidase.