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The focus on dopamine-sensitive motor symptoms, in association with the improvement of motor complications in the heterogeneous disease entity Parkinson's disease, has led to a certain standstill in research.
microRNAs --- disease-modifying effects --- SH-SY5Y --- inpatient treatment --- sphingolipids --- neurodegeneration --- magnetic resonance spectroscopy --- MO3.13 --- schizophrenia --- epigenetics --- long-chain acylcarnitine --- molecular mimicry --- curli --- multimodal complex treatment --- chlorogenic acid --- glycerophospholipids --- caffeic acid --- lipoproteins --- myenteric plexus --- neurometabolites --- HOG --- immature oligodendrocyte --- autophagy --- oligodendrocyte --- hypochlorite --- alpha-synuclein --- microbiome --- glucocerebrosidase --- Lewy Body Dementia --- brain iron --- mesenchymal stem cells --- fatty acid ?-oxidation --- GABA --- enteric glial cell --- gut-brain axis --- microbiota --- REM sleep behavior disorders --- redox cycling --- magnetic resonance imaging --- differentiation --- ?-synuclein-mediated pathology --- Parkinson disease --- Gaucher’s disease --- metallothionein --- [123I]FP-CIT-SPECT --- dopaminergic neuron --- mature oligodendrocyte --- glycerolipids --- spectroscopy --- neuroinflammation --- exosomes --- immunotherapy --- DNA methylation --- sterol lipids --- nigral cells --- cell line --- cysteinyl-dopamine --- myelin --- parkinsonisms --- multiple sclerosis --- rotenone --- brain --- fatty acyls --- Krabbe’s disease --- neuroprotection --- DAT --- motor dysfunction --- secretome --- multiprofessional therapy --- Parkinson’s disease --- oxidative stress --- alpha-Synuclein
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