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Somatostatin --- Immunohistochemistry. --- Immunohistochemistry. --- Somatostatin --- Analysis. --- Analysis.
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Pathological endocrinology --- Paediatrics --- Growth disorders. --- Somatomedin. --- Somatostatin. --- Somatotropin. --- Growth Disorders.
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Housing. --- Nest building. --- Nest. --- Oxytocin. --- Pig. --- Pregnancy. --- Progesterone. --- Prolactin. --- Prolactine. --- Reproduction. --- Somatostatin. --- Sow. --- Sows. --- Variation.
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Acromegaly --- Somatostatin --- drug therapy --- analogs & derivatives --- therapeutic use --- Octreotide acetate --- Chemotherapy --- Congresses. --- Drug therapy --- Therapeutic use --- Testing --- Analogs & derivatives
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Regurgitation is a way of feeding animals, usually young, by disgorging partially digested food. Except for a similar way of feeding in some species of birds, regurgitation is used exclusively by canids (as far as we know today). Earlier, it had been suggested that regurgitation was an important step in the weaning process. More recently, however, it has been shown that this behaviour no longer occurs in all domestic dogs. We review the literature about regurgitation behaviour and digestive and lactational physiology; all discussed with reference to weaning. We suggest that weaning may be influenced by the bitch giving the pups a chance to choose easily accessible regurgitated food instead of milk. This may lead to a smoother weaning process, with a lower level of aggression from the mother as a consequence. Furthermore, the physiological status of the pups during the weaning period might be influenced if the regurgitated, partly digested food, is important for their digestion. Mechanical preparation of the food or supply of gastrin, gastric juice and other components could help to improve digestion. Lastly, since the digestive and lactogenetic systems are activated simultaneously at both suckling and feeding, a decrease in both systems might cause a bitch to neither digest nor produce milk following ingestion; this would open the possibility for the food to be regurgitated
Aggression. --- Animal. --- Animals. --- Behavior. --- Behaviour. --- Bird. --- Boxes. --- Canids. --- Digestion. --- Dog. --- Dogs. --- Domestic dog. --- Feeding and nutrition. --- Feeding. --- Food. --- Gastrin. --- Insulin. --- Lactation. --- Level. --- Light. --- Milk. --- Mother. --- Old. --- Parent-offspring conflict. --- Physiological. --- Physiology. --- Pups. --- Ranging domestic pigs. --- Regurgitation. --- Release. --- Review. --- Somatostatin. --- Strategies. --- Strategy. --- System. --- Systems. --- Time. --- Weaning. --- Young.
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Considerable attention has been focused on the role of corticotropin releasing factor (CRF) as well as CRF-binding protein (CRF-BP) in neuropsychiatric disorders and neurodegenerative diseases including epilepsy. Therefore, in the present study, we investigated the temporal and spatial alteration of CRF and CRF-BP in the gerbil hippocampal complex in order to characterize the possible changes and associations with different sequelae of spontaneous seizure in these animals. CRF immunoreactivity was shown in the interneurons of the hippocampal complex at 30 min following seizure. Additionally, alteration of CRF-BP immunoreactivity was restricted to the entorhinal cortex after seizure. These results indicate some factors for consideration. First, in the gerbil hippocampal complex, the delayed increase of CRF immunoreactivity, in spite of its excitatory function, may attenuate seizure activity, but may not do so in epileptogenesis. Second, in contrast to the hippocampl complex, the increase in CRF-BP immunoreactivity in the entorhinal cortex following seizure may participate in feedback inhibitory modulation. (C) 2003 Elsevier Science Ltd. All rights reserved
Activity. --- Animal. --- Animals. --- Association. --- Attention. --- Behavioral-responses. --- Brain. --- Convulsive seizures. --- Cortex. --- Corticotropin-releasing-factor. --- Crf. --- Dentate gyrus. --- Disease. --- Diseases. --- Disorder. --- Entorhinal cortex. --- Epilepsies. --- Epilepsy. --- Expression. --- Feedback. --- Function. --- Gerbil. --- Hippocampal complex. --- Hippocampal. --- Hippocampus. --- Immunoreactive neurons. --- Immunoreactivity. --- Increase. --- Kainate-elicited seizures. --- Modulation. --- Mongolian gerbil. --- Protein. --- Rat. --- Seizure. --- Somatostatin. --- Spatial. --- Spontaneous seizure. --- Subiculum. --- Synaptic connections. --- Time.
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This book focuses on the recent advancements in both fundamental and clinical research, focusing on identifying, developing, and applying new and improved biological markers for specific neurologic disorders in the future. The original research work and review articles published here highlight some unique mechanisms underlying the most prevalent pathophysiological conditions affecting human health. Other areas covered in the book include emerging treatment options and correct diagnoses using different biochemical and imaging techniques.
Medicine --- Alzheimer’s disease --- amyloid beta --- hippocampus --- Kir channels --- K+ channels --- glioblastoma multiforme --- low-grade glioma --- astrocytoma --- recurrence --- multifocal growth --- mRNA expression --- MPS1 --- TTK --- therapy --- EDTA chelation therapy --- neurodegenerative diseases --- metal detoxification --- reactive oxygen species (ROS) --- neuroinflammation --- neurodegeneration --- cognitive dysfunction --- neurofibrillary tangles --- diagnosis methods --- biomarkers --- perinatal stress --- NURR1 --- inflammation --- late-onset diseases --- early life --- artificial neural network --- deep learning --- Parkinson’s disease --- atypical parkinsonian syndrome --- dopamine transporter SPECT --- β-Amyloid --- calpain --- Collapsin Response Mediator Protein-2 --- human-neuroblastoma SH-SY5Y cells --- Somatostatin-14 --- somatostatin receptor --- brain injury --- quinpirole --- dopamine D2 receptors --- glial cell --- autophagy --- mitophagy --- multiple sclerosis --- biomarker --- neurons --- astrocytes --- Alzheimer’s and Parkinson’s diseases --- fluid and imaging biomarkers --- amyloid-β and tau --- miRNA --- extracellular vesicles --- exosomes and ectosomes --- PET --- radiotracers --- radiolabeled molecules --- n/a --- Alzheimer's disease --- Parkinson's disease --- Alzheimer's and Parkinson's diseases
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Identification and development of cancer biomarkers and targets have greatly accelerated progress towards precision medicine in oncology. Studies of tumor biology have not only provided insights into the mechanisms underlying carcinogenesis, but also led to discovery of molecules that have been developed into cancer biomarkers and targets. Multi-platforms for molecular characterization of tumors using next-generation genomic sequencing, immunohistochemistry, in situ hybridization, and blood-based biopsies have greatly expanded the portfolio of potential biomarkers and targets. These cancer biomarkers have been developed for diagnosis, early detection, prognosis, and prediction of treatment response. The molecular targets have been exploited for anti-cancer therapy and delivery of therapeutic agents. This Special Issue of Biomedicines focuses on recent advances in the discovery, characterization, translation, and clinical application of cancer biomarkers and targets in malignant diseases of the digestive system. The goal is to stimulate basic and translational research and clinical collaboration in this exciting field with the hope of developing strategies for prevention and early detection/diagnosis of cancer in digestive organs, and improving therapeutic and psychosocial outcomes in patients with these malignant diseases.
n/a --- liver graft injury --- HFE --- neurokinin --- chemotherapy --- intestinal tumors --- therapeutic targets --- biliary tract carcinoma --- hepatocellular carcinoma --- clinical trial --- cell adhesion molecules --- colorectal cancer --- biomarkers --- phenotypic mosaics --- gastrointestinal oncology --- Asian Cancer Research Group (ACRG) --- biomarker --- psychosocial support --- precision therapy --- pancreatic carcinoma --- precision medicine --- Liver transplantation --- predictive biomarkers --- CD274 --- cholecystokinin --- The Cancer Genome Atlas (TCGA) --- gastrin --- pembrolizumab --- immunotherapy --- gastrin-releasing peptide --- stereotactic body radiation therapy --- immunohistochemistry --- gastric carcinoma --- liver transplant --- CAM invasion assay --- intragraft gene expression profiles --- molecular profiling --- targeted therapy --- neurotensin --- intestinal disorder --- ramucirumab --- next-generation sequencing --- colorectal carcinoma --- tumor progenitor --- circulating tumor cells --- gastrointestinal malignancies --- bombesin --- trastuzumab --- somatostatin --- zebrafish --- G protein–coupled receptors --- G protein-coupled receptors
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Croissance [Facteurs de ] --- Facteurs de croissance --- Factoren [Groei] --- Groeifactoren --- Growth substances --- Production de viande --- Meat production --- Ruminant --- ruminants --- Oestrogène --- oestrogens --- Androgène --- androgens --- Tissu adipeux --- adipose tissue --- Castration --- castration --- Facteur de croissance --- Croissance compensatoire --- Compensatory growth --- Ionophore --- Somatostatine --- Somatostatin --- Somatotropine --- Somatotropin --- 636.06 --- 591.134 --- 636.2 --- 577.175.3/.7 --- Characteristics, constitution and special biological features of domestic animals (size, temperament, organic changes etc.) --- Large ruminants. Cattle, oxen --- Hormones of vertebrates --- 577.175.3/.7 Hormones of vertebrates --- 636.2 Large ruminants. Cattle, oxen --- 591.134 Growth --- 636.06 Characteristics, constitution and special biological features of domestic animals (size, temperament, organic changes etc.) --- 637.5 --- #ABIB:aeco --- 637.5 Meat. Flesh products for food --- Meat. Flesh products for food --- Growth --- genetics --- Metabolism --- Growth factors --- Ionophores --- Regulation --- Livestock --- castration. --- Promoteur de croissance --- Composition --- Meat research --- Transgenes --- Autocrine --- Paracrine --- Controle nutritionnel --- Igf
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