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This book provides new and in-depth insights into molecular aspects of plant cell signaling in response to biotic, such as aphid- and grey mold disease-resistance, and abiotic stresses, such as soil salinity and drought stress, and additionally, functional analysis on signaling components involved in flowering, juvenility, GA signaling, and biosynthesis, and miRNA-regulated gene expression. Furthermore, plant acclimation was reported, with emphasis on mechanistic insights into the roles of brassinosteroids, cyclic AMP, and hydrogen sulfide, and the recent advances of transmembrane receptor-like kinases were refined. Clearly, plant cell signaling is an intensive topic and whether it is now or in the future, the emerging technology in functional analysis such as genome editing technologies, high-throughput technologies, integrative multiple-omics as well as bioinformatics can assist researchers to reveal novel aspects of the regulatory mechanisms of plant growth and development, and acclimation to environmental and biotic stresses. The achievement of such research will be useful in improving crop stress tolerances to increase agricultural productivity and sustainability for the food supply of the world.

Research & information: general --- Biology, life sciences --- salinity --- selenium (Se) --- crops --- reactive oxygen species (ROS) --- enzymatic anti-oxidative system --- drought --- GA --- DELLA --- ABF2 --- protein-protein interaction --- Arabidopsis --- endocytosis --- microRNAs --- miPEPs --- peptides --- development --- kinase --- receptor --- stress --- tobacco --- calcium --- calcite --- reactive oxygen species --- ion channels --- cellular signalization --- brassinosteroids --- receptor-like kinases --- GSK3-like kinases --- somatic embryogenesis receptor-like kinases --- protein phosphatases --- Malus domestica --- Rosaceae --- juvenility --- FLOWERING LOCUS C --- flowering --- Hydrogen sulfide --- S-sulfhydration --- plant hormone --- gasotransmitter --- disease resistance --- plant defense --- herbivore --- phytohormone --- plant biotic stress --- plant signalling --- Medicago truncatula --- abiotic stress --- cAMP --- cyclic nucleotides-gated channels --- plant innate immunity --- Botrytis cinerea --- tomato --- iprodione --- mutant --- transcriptome analysis --- metabolism --- catalytic activity --- dwarfism --- gene cloning --- MNP1 --- CPS --- ABA signaling --- brassinosteroid signaling cascade --- drought tolerance --- priming --- stress adaptation --- stress memory --- CRISPR/Cas9 --- DELLA/TVHYNP --- Dwarf --- GA20OX2 --- GA signaling --- salinity --- selenium (Se) --- crops --- reactive oxygen species (ROS) --- enzymatic anti-oxidative system --- drought --- GA --- DELLA --- ABF2 --- protein-protein interaction --- Arabidopsis --- endocytosis --- microRNAs --- miPEPs --- peptides --- development --- kinase --- receptor --- stress --- tobacco --- calcium --- calcite --- reactive oxygen species --- ion channels --- cellular signalization --- brassinosteroids --- receptor-like kinases --- GSK3-like kinases --- somatic embryogenesis receptor-like kinases --- protein phosphatases --- Malus domestica --- Rosaceae --- juvenility --- FLOWERING LOCUS C --- flowering --- Hydrogen sulfide --- S-sulfhydration --- plant hormone --- gasotransmitter --- disease resistance --- plant defense --- herbivore --- phytohormone --- plant biotic stress --- plant signalling --- Medicago truncatula --- abiotic stress --- cAMP --- cyclic nucleotides-gated channels --- plant innate immunity --- Botrytis cinerea --- tomato --- iprodione --- mutant --- transcriptome analysis --- metabolism --- catalytic activity --- dwarfism --- gene cloning --- MNP1 --- CPS --- ABA signaling --- brassinosteroid signaling cascade --- drought tolerance --- priming --- stress adaptation --- stress memory --- CRISPR/Cas9 --- DELLA/TVHYNP --- Dwarf --- GA20OX2 --- GA signaling

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Acute kidney injury (AKI) is still associated with high morbidity and mortality incidence rates, and also bears an elevated risk of subsequent chronic kidney disease. Although the kidney has a remarkable capacity for regeneration after injury and may recover completely depending on the type of renal lesions, the options for clinical intervention are restricted to fluid management and extracorporeal kidney support. The development of novel therapies to prevent AKI, to improve renal regeneration capacity after AKI, and to preserve renal function is urgently needed. The Special Issue covers research articles that investigated the molecular mechanisms of inflammation and injury during different renal pathologies, renal regeneration, diagnostics using new biomarkers, and the effects of different stimuli like medication or bacterial components on isolated renal cells or in vivo models. The Special Issue contains important reviews that consider the current knowledge of cell death and regeneration, inflammation, and the molecular mechanisms of kidney diseases. In addition, the potential of cell-based therapy approaches that use mesenchymal stromal/stem cells or their derivates is summarized. This edition is complemented by reviews that deal with the current data situation on other specific topics like diabetes and diabetic nephropathy or new therapeutic targets.

microRNAs --- n/a --- transcription --- ischemia/reperfusion injury --- DSS-colitis --- kidney inflammation --- therapeutics targets --- CXCL13 --- glomerulus --- interleukin-6 --- rhabdomyolysis --- IgA nephropathy --- CREB Regulated Transcriptional Coactivators (CRTC) --- slit diaphragm --- injury --- xanthine oxidase --- Salt Inducible Kinase (SIK) --- acute and chronic kidney disease --- therapeutic target --- KIT-IgA score --- G-protein-coupled bile acid receptor (TGR5) --- lysophosphatidic acid --- glomerular injury --- IL-18 --- mesenchymal stem cells --- Taiwan --- acute kidney injury --- renal ischemia-reperfusion --- long non-coding RNA --- fibrosis --- acute kidney failure --- diabetic kidney diseases --- chronic kidney disease --- lncRNA --- LPS-binding protein --- endotoxemia-induced oliguric kidney injury --- dapagliflozin --- cPLA2 and COX-2 --- NLRP3 inflammasome --- CmklR1 --- haem --- chronic kidney injury --- omega-3 fatty acid --- noninvasive --- inflammation --- regulated necrosis --- GLP-1 receptor agonists --- miRNA --- AKI --- SGLT2 inhibitors --- diabetic kidney disease --- extracellular vesicles --- podocin --- type IV collagen --- epithelial cells --- nephrin --- 2-kidney-1-clip --- renal fibrosis --- papilla --- diagnostics --- necrosis --- non-coding RNAs --- podocyte --- Thy1.1 nephritis --- KIT assay --- oxidative stress --- conditioned medium --- C-reactive protein --- pericyte --- myofibroblast --- Nuclear Factor kappa-light-chain-enhancer of activated B cells (NF-?B) --- endotoxemia --- modifier gene --- polymorphism --- renal stem cells --- kidney --- polyploidization --- Class IIa Histone Deacetylases (HDAC) --- 2k1c --- molecular signaling --- proximal tubule --- arachidonic acid --- empagliflozin --- tubular injury --- signaling cascade --- signal transduction --- inflammatory maker --- niches --- biomarkers --- renal progenitors --- type V collagen --- cyclooxygenase --- focal segmental glomerulosclerosis --- inflammatory bowel disease (IBD) --- chronic kidney disease (CKD) --- allograft rejection --- renovascular hypertension --- genotype --- molecular mechanisms --- ROS --- prediction --- glomerular filtration barrier (GFB) --- alport syndrome --- scattered tubular cells --- long non-coding RNAs --- renal inflammation --- lysophosphatidic acid receptor --- cAMP Regulatory Element Binding Protein (CREB) --- Farnesiferol B --- differentiation --- mesenchymal stromal cells --- modified-MSCs --- kidney transplantation --- polyunsaturated fatty acids --- apoptosis --- type I collagen --- diabetes mellitus --- natural products --- lipoxygenase --- stem cell --- T cell-mediated rejection --- exosomes --- renal injury --- obese kidney fibrosis --- kidney injury --- cytotoxicity --- mesenchymal stem cell --- pigment nephropathy --- mesodermal stem cell --- ischemia-reperfusion --- cytochrome P450 --- renal cell carcinoma --- hematuria --- B-cell attracting chemokine --- microRNA --- chemerin --- glomerular basement membrane --- glomerular damage --- renal tubular cells --- kidney proximal tubule --- exosome --- hypertension --- diabetic nephropathy