Export directly to RefWorks

TY  - BOOK
ID  - 145255356
TI  - New Insights into the Pathogenesis and Therapies of IgA Nephropathy
AU  - Suzuki, Hitoshi
AU  - Novak, Jan
PY  - 2022
PB  - Basel MDPI Books
DB  - UniCat
KW  - Medicine
KW  - obesity
KW  - mesangial matrix expansion
KW  - body mass index
KW  - IgA nephropathy
KW  - IgA Vasculitis
KW  - IgA Nephropathy
KW  - adults
KW  - children
KW  - presentation
KW  - physiopathology
KW  - genetics
KW  - prognosis
KW  - treatment
KW  - IgA
KW  - clinical trials
KW  - kidney mesangium
KW  - mouse model
KW  - aberrantly glycosylated IgA1
KW  - galactose-deficient IgA1
KW  - glycosylation of IgA1
KW  - biomarker
KW  - complement C3
KW  - O-glycosylation
KW  - IgA1
KW  - autoantibody
KW  - immune complex
KW  - complement
KW  - kidney
KW  - nephrology
KW  - IgA vasculitis
KW  - nephritis
KW  - kidney biopsy
KW  - plasma cells
KW  - CD38
KW  - renal pathology
KW  - urinary galactose-deficient IgA1
KW  - KM55
KW  - crescents
KW  - proteinuria
KW  - glomerular filtration rate
KW  - Oxford score
KW  - obesity
KW  - mesangial matrix expansion
KW  - body mass index
KW  - IgA nephropathy
KW  - IgA Vasculitis
KW  - IgA Nephropathy
KW  - adults
KW  - children
KW  - presentation
KW  - physiopathology
KW  - genetics
KW  - prognosis
KW  - treatment
KW  - IgA
KW  - clinical trials
KW  - kidney mesangium
KW  - mouse model
KW  - aberrantly glycosylated IgA1
KW  - galactose-deficient IgA1
KW  - glycosylation of IgA1
KW  - biomarker
KW  - complement C3
KW  - O-glycosylation
KW  - IgA1
KW  - autoantibody
KW  - immune complex
KW  - complement
KW  - kidney
KW  - nephrology
KW  - IgA vasculitis
KW  - nephritis
KW  - kidney biopsy
KW  - plasma cells
KW  - CD38
KW  - renal pathology
KW  - urinary galactose-deficient IgA1
KW  - KM55
KW  - crescents
KW  - proteinuria
KW  - glomerular filtration rate
KW  - Oxford score
UR  - https://www.unicat.be/uniCat?func=search&query=sysid:145255356
AB  - IgA nephropathy (IgAN) is the most common form of primary glomerulonephritis worldwide and a frequent cause of kidney failure. Better understanding of the pathogenesis of IgAN and the related genetic, immunological, and cellular susceptibility factors are needed to enable the development of effective disease-specific therapy. This book brings together international experts to provide clinical and experimental studies and reviews with an emphasis on early diagnosis, prognosis, disease pathogenesis, determination of disease activity, and new strategies for treatment for IgAN.
ER  -